The stroke, which left the Southwestern Research Institute (SWRI) director in a coma and forced the institution to shut down in 2013, was the first time that a person with a stroke had ever suffered brain damage.
The event has also led to a series of studies in which researchers have attempted to determine what happens in the brain after a stroke.
SWRI’s study, published in the journal Brain, was funded by the National Institutes of Health (NIH).
SWRI researcher Chris Smith and colleagues wanted to understand how the stroke affected the brain’s ability to generate new neurons, or synapses, that can be used by brain cells to communicate.
In their study, Smith and his colleagues used a modified version of a magnetic resonance imaging (MRI) scanner called a magnetoencephalography (MEG) to track activity in the brains of patients who had suffered a stroke, as well as a control group of healthy volunteers.
The study was designed to assess how the brain responded to a stroke and its effects on neural function, the researchers said.
After a stroke patients have an increased risk of cognitive impairment.
Smith said the results of the study showed that patients with stroke had significantly less neural activity in their brains compared to the control group.
In addition, brain activity in patients with a brain injury was more similar to the brains activity in healthy controls than to the brain activity of stroke patients.
“We found that stroke patients had a significantly lower percentage of neural activity compared to healthy controls,” Smith said.
“Our data suggest that stroke affects neural function in a way that leads to cognitive impairment, which is consistent with our prior findings.”
This means the stroke patients’ brains are less active, and less efficient at generating new neurons to connect with other neurons.
This may be because the stroke’s disruption of the brain can have effects on how the body processes information, Smith said, adding that the study supports the theory that the brain could become less efficient with age.
Brain imaging research is often used to better understand the brains and behavior of people with stroke, and it has also shown that stroke is associated with increased risk for dementia.
“These results suggest that the brains are not more efficient at using new neurons for communication,” Smith wrote.
“However, the study does not provide clear evidence for the role of a brain network as a key to dementia.”
For the study, researchers recruited healthy volunteers who had been admitted to the SWRI for a stroke in the previous year.
The volunteers underwent two brain scans.
One was performed while they were lying down, and the other was performed before they were taken to the hospital for treatment.
The scans showed that the participants’ brains were in a constant state of electrical activity, meaning that the neurons in the stroke participants’ brain cells were continually firing.
The researchers then scanned the brains while they performed a mental arithmetic task.
In both scans, brain waves showed activity in two regions of the participants brains: the left hippocampus and the right prefrontal cortex.
The left hippocampus was more active than the right hippocampus.
This indicates that the left prefrontal cortex was more involved in processing information than the left hippocampal region.
The right prefrontal region is the area of the left brain that is active during memory and learning.
The activity of this region can also be observed in people with dementia, including people who have Alzheimer’s disease.
This is important because the activity of the right and left prefrontal regions may help to improve memory.
The findings were then compared to a control condition in which no stroke was present.
The participants were then given an MRI scan.
The scan showed that their brains were no longer in a state of continuous electrical activity.
The brains activity was much less active than in the MRI scan, indicating that the stroke had altered their brain’s electrical properties.
“The results indicate that the patients’ brain was not fully responding to electrical stimulation,” Smith concluded.
“This indicates that neuronal activity is less efficient than in people who are healthy and normal.
The results also indicate that patients who have stroke have lower brain function.”
The study appears in the Journal of Neuroscience.
In a second study, the team also used MRI to scan the brains, but the patients had undergone a stroke as well.
This was done in the same way as the MRI scans, and showed a similar pattern of decreased activity in brain regions of stroke.
The difference between the MRI and MRI scans was that the researchers used a new technology called positron emission tomography (PET), which uses light to detect changes in the electrical properties of a person’s brain.
The positron emissions of these devices are used to detect electrical activity in people’s brains that is not present in people without stroke, because those with stroke can’t produce the same electrical activity as healthy people.
“PET is a better and more accurate way to look at brain activity, but it has limitations,” said lead author of the new study, Christopher Smith, a postdoctoral fellow in neuroscience at the University of Utah.
“In a stroke